If treatment stops, symptoms often reappear, especially if they have already returned after previously stopping medications.
According to the National Institute of Mental Health researchers, people who have a close relative with schizophrenia are more likely to develop the disorder than are people who have no relatives with the illness. Schizophrenia Research,e Recent research has found that these genetic differences involve hundreds of different genes and gene mutations, and probably disrupt brain development.
The reduced transcription of synaptic proteins suggests that schizophrenia is a disorder of neuronal miswiring. Treatment must continue, even when symptoms seem to have receded. Genome wide linkage studies have identified at least 15 susceptible genetic loci with weak to moderate linkages to schizophrenia including DISC1, Neuregulin 1, Dysbindin, D-amino acid oxidase activator and catechol-o-methyl transferase.
It appears that many but probably not all of these changes are present before an individual becomes ill, and schizophrenia may be, in part, a disorder in brain development. Caregivers and family members can help the person who has a diagnosis by learning as much as possible about schizophrenia and by encouraging the patient to adhere to their treatment plan.
Environmental Triggers If you were exposed to certain viral infections before you were born, there is some rersearch to suggest that possibly your chances of getting schizophrenia may be higher. These are both possible theories, but they have not been conclusively proven by scientific studies.
Researchers Etiology of schizophrenia essay to examine the genetic factors and gene sets connected to schizophrenia. Scientists think that in these cases, a gene may have changed and made the condition more likely.
Scientists think interactions between genes and the environment are necessary for schizophrenia to develop. The more they learn, the more it becomes evident that the condition is far more complex than anyone realized.
These faulty connections may lie dormant until puberty. The brains of people with the illness also tend to have less gray matter, and some areas of the brain may have less or more activity. Magnetic resonance spectroscopy MRS of schizophrenics showed glutamatergic abnormalities due to decreased levels of the amino acid N-acetyl aspartic acid NAA in the prefrontal cortex, temporal cortex, thalamus, basal ganglia, and cerebellum.
Support can include improving communication skills, finding work and housing, and joining a support group. Research shows it takes a combination of genetics and your environment to trigger the disease.
It appears that areas of specific genes, gene mutations, and certain sets of genes are implicated in schizophrenia. Positive or psychotic symptoms include delusions, hallucinations, thought disorder, and abnormal psychomotor activity.
Genetics Think of your genes as a blueprint for your body. Side effects may include short-term memory loss. Season of birth, geographical variation, substance abuse, uterine environment, prenatal stress, prenatal exposure to environmental toxicants, family size, and availability of adequate maternal diet are contributing factors in the development of schizophrenia.
Some research has shown that marijuana use has a similar risk. Praveen Sharma, in Biomarkers in ToxicologySchizophrenia The unknown etiology of schizophrenia makes it a very complex psychiatric disorder, affecting 0.
Hospitalization can help people get back onto their medication while keeping them safe. Spaces in the brain, called ventricles, were larger.
The seizure is thought to trigger a massive neurochemical release in the brain. Due to the advances in neuroimaging and our understanding of schizophrenia, researchers can study the functioning brain as it carries out activities this is called functional magnetic resonance imaging.
Cognitive symptoms include disturbances in selective attention, working memory, executive control, episodic memory, language comprehension, and social emotional processing. It can take some time to reach a diagnosis. A monozygotic identical twin of a person with schizophrenia has the highest risk — a 40 to 65 percent chance of developing the illness.
There is no known single cause of schizophrenia. Changes in neural substrates, such as AMPA and NMDA receptors and their affiliated signaling, trafficking, scaffolding, and anchoring proteins impact learning, memory, and other cognitive functions that underlie neuroplasticity.
Retrieved on September 7,from https:What causes schizophrenia? Learn more about the genes, environmental factors, and brain changes that contribute to this mental disorder. The causes of schizophrenia, like all mental disorders, are not completely understood or known at this time. Researchers have spent millions of hours (and many hundreds of millions of dollars.
to account for the etiology of schizophrenia. These two models, which are antecedents of the different developmental perspectives, explain how these perspectives differ and how they can be applied to gain better knowledge of the etiology of psychopathology.
Schizophrenia Essay Words | 11 Pages. Schizophrenia Schizophrenia is a serious. The purpose of this paper is to explore schizophrenia, in particular its signs, symptoms, etiology and pathophysiology, and then examine drug treatment and their mechanism of action.
This will be followed by the prognosis for people with schizophrenia.
People with schizophrenia often experience fear, confusion, and paranoia. It is a chronic mental health condition that affects a person's thought processes, making it difficult to think clearly. Probing the etiology of schizophrenia currently relies upon brain imaging, postmortem, and animal studies, although a wealth of information has been garnered using such methodologies, there are limitations.
Brain imaging studies reveal information about brain volume and structural changes at the gross level only; postmortem studies uncover the.Download